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Blacks with the mutation ended up with LDL levels averaging 100 instead of the usual 138, a 28 reduction; whites with a less powerful mutation had LDL levels averaging 117, about 15 lower than average. The people with one disabled gene had lower than normal LDL levels for their entire lives. Hobbs and Cohen found that blacks aged 45 to 64 with a single mutated gene seemed almost immune to coronary heart disease during a 15-year follow-up, and whites, who had the less powerful mutation, had a 46 reduction in the incidence of coronary heart disease. These findings led them to search for a mother and father who each carried the single mutated gene.

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